Cristin-resultat-ID: 1284232
Sist endret: 25. oktober 2016 14:34
Resultat
Vitenskapelig foredrag
2015

Borderline and antisocial personality disorder traits in early to middle adulthood: A longitudinal twin study

Bidragsytere:
  • Ted Reichborn-Kjennerud
  • Nikolai Olavi Czajkowski
  • Eivind Ystrøm
  • Ragnhild Ørstavik
  • Steven H. Aggen
  • Kristian Tambs
  • mfl.

Konferanse

Tittel: ISSPD - Personality Disorders Across the Lifespan - October 13-16 2015

Sted:

Montreal
Canada
Utbredelsesområde: Internasjonalt
Årstall: 2015
Dato fra: 13. oktober 2015
Dato til: 16. oktober 2015

Arrangør:

The International Society for the Study of Personality Disor

Om resultatet

Vitenskapelig foredrag
Publiseringsår: 2015
Invitert
Fagfellevurdert
Plenar

Beskrivelse Beskrivelse

Tittel

Borderline and antisocial personality disorder traits in early to middle adulthood: A longitudinal twin study

Sammendrag

Background and Aims: Antisocial personality disorder (ASPD) and borderline personality disorder (BPD) share genetic and environmental risk factors. Little is known about the temporal stability of these etiological factors in adulthood. Methods: DSM-IV criteria for ASPD and BPD were assessed using structured interviews in 2,282 Norwegian twins in early adulthood and approximately 10 years later. Longitudinal biometric models were used to analyze the number of endorsed criteria. Results: The mean criterion count for ASPD and BPD decreased 40% and 28% respectively from early to middle adulthood. Rank-order stability was 0.58 for ASPD and 0.45 for BPD. The best-fitting longitudinal twin model included only genetic and individual-specific environmental factors. Genetic effects, both those shared by ASPD and BPD, and those specific to each disorder remained completely stable. The unique environmental effects, however, changed substantially, with a correlation across time of 0.19 for the shared effects, and 0.39 and 0.15 respectively for those specific to ASPD and BPD. Genetic effects accounted for 71% and 72% of the stability over time for ASPD and BPD respectively. The genetic and environmental correlations between ASPD and BPD were 0.73, and 0.43 respectively at both time points. Conclusion: ASPD and BPD traits were moderately stable from early to middle adulthood, mostly due to genetic risk factors which did not change over the10 year assessment period. Environmental risk factors were mostly transient, and appear to be the main source of phenotypic change. Genetic liability factors were, to a large extent, shared by ASPD and BPD.

Bidragsytere

Ted Reichborn-Kjennerud

  • Tilknyttet:
    Forfatter
    ved Enhet voksenpsykiatri ved Universitetet i Oslo

Nikolai Olavi Czajkowski

  • Tilknyttet:
    Forfatter
    ved Psykologisk institutt ved Universitetet i Oslo

Eivind Ystrøm

  • Tilknyttet:
    Forfatter
    ved Galenisk farmasi og samfunnsfarmasi ved Universitetet i Oslo

Ragnhild Elise Ørstavik

Bidragsyterens navn vises på dette resultatet som Ragnhild Ørstavik
  • Tilknyttet:
    Forfatter
    ved Avdeling for kroniske sykdommer og aldring ved Folkehelseinstituttet

Steven H. Aggen

  • Tilknyttet:
    Forfatter
1 - 5 av 12 | Neste | Siste »